The Never Ending Questions on Spike Protein - Sub Unit 1 Concerns
So I got this from Jessica Rose substack. I wanted to highlight it, because it answers a question that I have been asking for ages (and then goes on to raise even more questions). And that on one, that until now, no one has been able to answer.
What is confirmed is that as soon as the Spike attaches itself to ACE2, the remaining S1 sub units do cleave off, and then can float freely around the body. This has MASSIVE IMPLICATIONS in my opinion for ALL JABS. Human bodies are being FLOODED with S1 and these will end up everywhere, and all over the brain neurological networks. "Because of the furin-cleavage site, the spike protein once bound to ACE-2, is cleaved and this yields 3 free S1 subunits per spike trimer, able to impose whatever subsequent damage they may. For someone with a high viral replication rate and high viral load, this will necessarily imply more free spike."
I have also attached Richard Flemmings tweet for thought. He and McCairn are following spike deaths closely. The Immune System is also NOT clearing the full LNP/SP as fast as thought. In fact, I would argue that it is not clearing full SP for a period allot longer than imagined, as they are not testing for this. Which leads into the question of circulating S1 sub unit, and how many months or years will these be present. The more and longer they are circulating, the more likely a poor outcome. And in light of thoughts on GoF, the Spike is the GoF tool. It is also what is being used in the jabs (a highly modified unstable optimised version to the wild Spike at that). So my argument that the real pandemic only started when the first needle burst into an arm, carries more weight. Natural infection does not have the viral load or Spike production that jabs do. There is also NO RATIONAL reason for them to be still using Wuhan 1 or any spike to be realistic - its a futile game of catch up and making things worse (its pure malevolence)
https://www.mdpi.com/2227-9059/10/7/1538
(edited)
Some Average Dumb Guy follow up questions to the above. Are these answerable? If so, please be kind and patient enough to indulge me.
Free floating S1 and the gut; a pathway to the brain? I asked this question a very long time ago. It went hand in hand with "does stomach acid destroy Spike"? I also asked how long these proteins would last in nature before being naturally degraded down. Which led to me asking how does "malfunctioning RNA" work in transfection? Will it still produce a spike of sort (also with regards to degraded payload packages in LNP).
There was research done on Newts and spike ages ago. It showed them being completely fucked up by spike in water. When I asked "what about spike we excrete into the ecosystem" it got shot down. Which then all leads me to "how much spike is lethal"? Can one folded protein (exome) touching the "right" part of the brain kick off CJD?
How many S1 sub units will it take to cause damage to key operational neurological centres?
This is how my mind works.
If any spike, or sub units are in general circulation (internal and external) what and how much is a problem? This is NOT a natural pathogen from nature. So it needs to be treated and viewed with GREAT SCRUTINY. If it was natural I would not be so overly cautious about it. But its not, and they are injecting it into peoples bodies.
And the "cleaving off of all sub units at time of "docking" onto host cell, just blows all theories of immune system coping for me! The magnitude of SP through jab versus natural infection are insane! And this also then shows us why the vector jabs are also indicating mass injuries ...not so?
And "shedding" I think this needs to be looked at thoroughly. If a jabbed person can exhale viral RNA & exomes. Then they could exhale S1 sub unit. How long would it take for S1 to degrade? If in a room packed with shedding jabbed folk, how much could we inhale or ingest? Which leads me back to the questions raised in my previous post ie what is a lethal or detrimental exposure to S1 sub unit?
These are questions that simply cannot be ignored or dismissed. They need a definitive answer. I know that some publications have circulated on this. But in light of the emerging data, there is simply not enough data collection of S1 prevalence to write this off yet.
The fact is
that NOBODY actually knows what is going to happen, what the actual facts are, and are all still working blindly, with little pieces of the puzzle being slowly pulled together. The smartest of smart scientists and biologists cannot give ANY definite answers yet. So we cannot dismiss anything. And IMO all cards on still on the table